Scientists at the University of Adelaide in Australia have proposed a new mechanism for the treatment of multiple sclerosis. A study conducted by PhD aimed to understand specific enzymes in the cellular immune system, modulating the activation and migration of immune cells.
Together with other scientists' research has focused on the molecular PI3Kgamma, which involves the activation and movement of blood cells. "The worldwide celebration of PI3Kgamma is of interest in other inflammatory diseases such as diabetes, rheumatoid arthritis, and the development of molecular binding in our research."
Scientists have shown that the molecule is the appearance of experimental autoimmune encephalitis in laboratory animals, which is key to the development of research in the standard laboratory system framework. Gene changes, "delete" this particular molecule, give a high resistance to the development and protection of the nervous system, which is characterized by multiple sclerosis.
When the molecule is present, there is a serious damage to the central nervous system, leading to inflammation of the myelin sheath and loss of the myelin sheath in the brain. After this, the use of the results of an expert panel of oral actives that blocked the first signs of activity of the molecule PI3Kgamma's disease. This substance inhibits the development of clinical symptoms that even "cancel" the disease.
Our research results so far have been very promising. By blocking PI3Kgamma, we can reduce the activation of autoreactive cells in the immune system, reducing the emission of inflammatory molecules from the cellular immune system, leading to a sharp decline in the movement of immune cells in the central nervous system.
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